Metabolic Oncology · Virology · Immunology

Where Discovery
Becomes Therapy.

Tavargenix was founded on a scientific observation that challenged convention. Today, we translate the biology of TKTL1 into therapeutic development programs.


A signal no one
was looking for.

Science rarely announces its breakthroughs. More often, they arrive as a quiet inconsistency — a number that doesn't add up, a pattern that shouldn't be there.

The Observation

Something didn't fit.

Studying cancer cell metabolism, Dr. Johannes Coy noticed that certain tumor cells were processing glucose through an unexpected pathway — not a mutation, not an error, but a deliberate biological strategy for survival and unchecked growth.

The Question

What if metabolism was the real target?

While the scientific mainstream focused on genomics, Dr. Coy followed a different thread: the enzyme at the center of this metabolic shift. He named it TKTL1 — and spent years making the case that disrupting it could matter clinically.

The Commitment

From insight to institution.

Tavargenix was founded to do what individual scientists cannot: protect a discovery, fund its translation, and carry it through the long road from hypothesis to therapy.

"The data kept pointing somewhere no one wanted to look. Most researchers moved on. I stayed."
— Dr. Johannes Coy, Founder, Tavargenix

Why this still matters

At a time when cancer research focused almost exclusively on genetic mutations, the discovery of TKTL1 opened an entirely new dimension: metabolic reprogramming as a therapeutic target. Today, that once-dismissed hypothesis has become a recognised field of oncology research.

See the development pipeline →

Metabolism is not background noise.
It is strategy.

Rapidly proliferating cells require continuous production of ribose and acetyl-CoA — essential components for DNA synthesis and growth. TKTL1 appears to play a central role in enabling this metabolic advantage.

Understand the mechanism →
01

Metabolic Reprogramming

Research suggests TKTL1 alters how cells redirect glucose-derived metabolites, enabling sustained proliferation even under conditions that would halt normal cell growth.

02

Supply Line Modulation

If the metabolic supply lines that fuel rapid cell division can be selectively disrupted, disease progression may be meaningfully slowed — a hypothesis now supported by preclinical evidence.

03

Cross-Indication Relevance

Metabolic dependencies are not unique to cancer. Viral replication and immune dysregulation may rely on related mechanisms, opening doors beyond oncology.

04

Small-Molecule Approach

Tavargenix developed B-OT, a small molecule designed to modulate the TKTL1-associated pathway — progressed through preclinical work and Phase I safety assessment.


Discovery must
lead somewhere.

Scientific insight alone does not change patient outcomes. Translation does.

The lead program B-OT has progressed through preclinical development and completed a Phase I safety assessment. Further clinical planning is ongoing in selected indications. Indication-specific programs may be licensed into dedicated entities to ensure focused development and regulatory clarity.

B-OT Development Status

PreclinicalComplete
Phase I SafetyComplete
Phase II PlanningOngoing
Phase II / IIIPlanned
Program 01
Oncology

Metabolic modulation in proliferative disease contexts. Targeting TKTL1-associated pathways in tumor metabolism.

Clinical development planning
Program 02
Virology

Exploring host-cell metabolic dependencies in viral replication — a potential new front for TKTL1-related research.

Development partnerships
Program 03
Global Health

Early-stage exploration in underserved indication spaces with high metabolic disease burden.

Early exploration

Portrait
Dr. Johannes Coy

Foto einsetzen

Scientific persistence.

Dr. Johannes Coy did not set out to build a company. He set out to understand something that didn't make sense — and refused to stop when the field told him to move on.

His work on TKTL1 was met with skepticism in its early years, as paradigm-shifting science often is. Years later, metabolic reprogramming has become one of the most active areas in cancer biology. Tavargenix was built to carry that work forward — with rigor, patience, and a long-term view.

TKTL1
Discovery of the transketolase-like 1 gene's role in tumor metabolism
B-OT
Lead compound through preclinical development and Phase I
3+
Therapeutic areas under active development or exploration

Translational science requires collaboration.

We seek partners who share our commitment to rigorous science and long-term therapeutic development.

  • Clinical development partners
  • Co-development collaborations
  • Licensing discussions
  • Strategic investors

Tavargenix GmbH

Gräfenhäuser Str. 26, 64293 Darmstadt, Germany

info@tavargenix.com

+49 (0) 6151 / 365 31 0

Get in touch

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